Photobiomodulation · Mitochondrial Health · 670nm Research

Why We Built
670nm Into
Our Lights.

And you don't even know it. Here's the wavelength, the mechanism, the research — and why we built it into every Mitorion product.

670 nm CcO enzyme ↑ ATP more energy ↓ stress ↑ repair mitochondria — cytochrome c oxidase

You eat better.
You move more.
Something still
feels off.

Energy is inconsistent. Recovery is slower. Focus drops faster than it used to. Most people look at food. Some look at stress. Almost nobody looks at light.

But here's the uncomfortable truth: your cells run on signals. And one of the strongest signals your cells were built to receive — one that's almost entirely absent from modern indoor environments — is a specific type of light.

"We've simplified light into one thing: brightness. More light equals better, less light equals worse. That's not how your biology sees it."

Different wavelengths do different jobs. Blue light wakes you up — sharpens attention, tells your brain it's daytime. But there's another side most people never think about. Red and near-infrared light. And this is where things get interesting.

Around 670nm:
the wavelength that
builds you back.

You don't see 670nm light clearly. It sits at the deep red end of the visible spectrum — visible only as a dim red glow. But your body feels it. Because research shows this wavelength interacts directly with the machinery that produces energy inside your cells.

The 670nm Mechanism — From Photon to Energy (Karu, 2008)

670nm photon enters the cell

Red light at 670nm penetrates biological tissue more deeply than shorter wavelengths. It passes through the skin surface and reaches mitochondria inside the cell — the power plants where energy is made. Shorter wavelengths scatter; 670nm reaches its target.

CcO

Absorbed by cytochrome c oxidase

Inside the mitochondria is an enzyme called cytochrome c oxidase (CcO). Think of it as a gatekeeper for energy production — the last step in the electron transport chain before ATP is made. CcO contains chromophores that absorb light in the red and near-infrared range. 670nm sits directly on one of its absorption peaks.

ATP production becomes more efficient

When CcO absorbs 670nm photons, its electron transfer rate improves. The mitochondrial membrane potential stabilises. More protons flow through ATP synthase. The result: more ATP produced from the same substrate — like tuning an engine to run more cleanly on the same fuel. Karu's research established this mechanism across multiple cell types.

Cellular stress becomes easier to manage

With more ATP available, cells handle oxidative stress more efficiently. Repair processes that were deprioritised due to energy shortage resume. Reactive oxygen species are better managed. The overall cellular environment shifts from stressed to stable — not through chemical supplementation but through improved energy supply to the systems that already handle these problems.

This is not theory. These mechanisms have been observed in laboratory and clinical settings across multiple research teams over decades. The photobiomodulation effect of red and near-infrared light on mitochondrial function is one of the most replicated findings in modern light biology.

What the research
actually found.

Four independent research lines — different teams, different designs, different endpoints — all pointing in the same direction.

Blood glucose reduced Powner & Jeffery (2024) showed that 670nm light stimulation of mitochondria reduced blood glucose levels — suggesting that improved mitochondrial efficiency directly influences how the body processes circulating carbohydrates.
colour
contrast
Retinal function improved Shinhmar et al. (2021) found that a single exposure to 670nm light improved colour contrast sensitivity for a full week — associated with measurable mitochondrial rejuvenation in retinal cells. One exposure. Seven days of effect.
Mitochondrial gene expression Sommer (2019) documented that 670nm laser light altered mitochondrial gene expression — suggesting effects beyond immediate energy production to longer-term adaptive changes in how mitochondria are built and maintained.

The fruit fly data — and what it means

One study exposed fruit flies to daily 670nm light and documented up to 80% increase in ATP production, reduced inflammation, improved mobility, and extended lifespan. These results are striking — and need honest framing.

Fruit flies are not humans. You cannot directly map these numbers to human outcomes. But fruit fly mitochondria operate on the same fundamental biochemistry as human mitochondria. The mechanism — cytochrome c oxidase absorbing red light, improving electron transfer, increasing ATP — is conserved across species. The direction of the effect is what matters. And that direction is consistent across organisms, cell types, and research teams.

"It's like revving an engine without ever maintaining it. Your cells are still running — but they're not getting the support they evolved to expect."

The spectrum
your indoor light
is missing.

Modern LED lighting was designed for efficiency and brightness — not for biology. It delivers the wavelengths that make spaces visible. What it leaves out are the wavelengths that support the systems running beneath your awareness.

670nm in Context — Where It Sits and Why Indoor Light Misses It
670nm Cytochrome c oxidase absorption peak — 660–670nm LED: near zero at this wavelength 380nm 450nm 670nm 780nm+ Intensity Indoor LED — blue peak, 670nm absent 670nm — mitochondrial response window

Standard LED lighting peaks in the blue range (around 420–460nm) and drops sharply in red and near-infrared. At 670nm — exactly where cytochrome c oxidase absorbs best — LED output is close to zero. Your mitochondria never receive the signal that, outdoors in natural light, would be present for hours every day.

Your environment is
doing two things
at once.

The modern light trap isn't just about having too much of one thing. It's a simultaneous problem on both ends of the spectrum.

Too much — wrong signal
"Blue light all day and night"
"Screens close to eyes for hours"
"Stimulation without recovery"
"Melatonin suppressed after sunset"
"System always 'on'"
Too little — missing signal
"No red / near-infrared indoors"
"670nm absent from LED spectrum"
"Mitochondria never receive repair signal"
"No full-spectrum outdoor light"
"Cells running without maintenance"

The retina — your eyes — is one of the most mitochondria-dense tissues in the body. It's also one of the first places the missing-signal problem shows up, because the energetic demand is so high. The same mitochondria that need 670nm support are working hardest to process the screen light you're staring at. One input is stressing them. The other input — the one that would support them — is absent.

What actually
helps.

Not hacks. Not extremes. Better signals. The inputs your mitochondria were built to receive, delivered consistently. Small and regular beats large and occasional.

01

Get real daylight — regularly

Natural light contains what indoor light doesn't. Even short outdoor exposure provides the full spectrum, including red and near-infrared wavelengths that penetrate tissue and reach mitochondria. This doesn't require hours — consistent short exposure daily is what builds the biological effect over time. Your mitochondria are not making up for missed days; they're responding to the signal they receive today.

02

Don't destroy your nights

Mitochondrial repair runs on the same circadian timing system as everything else. Blue light at night disrupts melatonin — and mitochondrial melatonin, produced locally inside the cell, is one of the primary antioxidants that protects the energy machinery overnight. Damage your sleep signal, and you also reduce the cellular protection your mitochondria depend on for overnight repair.

03

Bring back the missing wavelengths

You don't need to live outside. You need to recognise that your indoor environment is spectrally incomplete — and address that gap. Red and near-infrared sources at appropriate intensity, used consistently, reintroduce the mitochondrial support signal that modern lighting removes. The research is clear on what wavelength, what tissue targets, and what the mechanism is. The engineering challenge is delivering it in a practical, everyday form.

04

Consistency over intensity

Short, regular exposure is more effective than occasional high-intensity sessions. Mitochondria adapt to consistent signals — not single large doses. The Shinhmar retinal study is instructive: a single 670nm exposure improved colour contrast for a week. Regular exposure builds and maintains the biological effect. This is how photobiomodulation works in the research — consistent, moderate, targeted.

Why We Built 670nm Into Our Lights

Not because it's trending.
Because the mechanism is real.

When we designed Mitorion's lighting products, we went back to the research. Not to marketing categories. Not to what looked impressive on a spec sheet. To the mechanism: cytochrome c oxidase, its absorption spectrum, the ATP response documented by Karu, the retinal results from Shinhmar, the glucose data from Powner and Jeffery.

670nm is not arbitrary. It sits precisely on the absorption peak of the enzyme your mitochondria use to produce energy. It's the wavelength at which the photobiomodulation effect is most reproducible, most studied, and most mechanistically understood.

We built it in because your cells are running indoors, all day, under lighting that was designed without their biology in mind. Our lights are designed with it in mind. That's the entire difference.

Your body is not just chemical. It's electrical. It's responsive. It adapts to input. And light is one of the most powerful inputs you have.

The research
behind this.

Scientific References
Karu TI (2008) — Mitochondrial signaling in mammalian cells activated by red and near-IR radiation. Photochemistry and Photobiology, 84(5):1091–1099. Foundational mechanistic work establishing that red and near-infrared light is absorbed by cytochrome c oxidase in the mitochondrial electron transport chain, improving electron transfer efficiency and increasing ATP production across mammalian cell types.
Shinhmar H, Grewal M, Pearce-Walker J et al. (2021) — Weeklong improved colour contrast sensitivity after single 670 nm exposures associated with mitochondrial rejuvenation. Scientific Reports, 11:22872. Demonstrated that a single 3-minute exposure to 670nm light improved colour contrast sensitivity for one week in adults over 40 — associated with measurable improvements in mitochondrial function in retinal cells. Provided human clinical evidence of meaningful biological response to a single targeted 670nm exposure.
Powner MB & Jeffery G (2024) — Light stimulation of mitochondria reduces blood glucose levels. Journal of Biophotonics. Showed that 670nm mitochondrial stimulation reduced blood glucose levels, linking mitochondrial photobiomodulation to systemic metabolic effects — specifically the mechanism by which improved mitochondrial efficiency alters circulating carbohydrate demand.
Sommer AP (2019) — 670 nm laser light and mitochondrial gene expression. Annals of Translational Medicine, 7(Suppl 1):S4. Documented that 670nm laser light altered mitochondrial gene expression, indicating that the effects of red light on mitochondrial function extend beyond immediate energy changes to longer-term adaptive changes in mitochondrial biogenesis and maintenance.
↑ ATP

Your body adapts
to input.
Give it the right one.

Right now, most people are living under light that keeps them functioning — but doesn't fully support them. You don't notice it immediately. But your cells do. Every day.

670nm is not a trend. It's a mechanism. And we built it in for exactly that reason.

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